Myeloid cells include monocytesmacrophagesneutrophilsbasophilseosinophilserythrocytesand megakaryocytes to platelets. Lig4 deficiency in the mouse causes a progressive loss of HSCs during aging. It was used extensively in early studies, but is now considered to measure more mature progenitor or transit-amplifying cells rather than stem cells. Journal of Stem Cells.
A cobblestone area-forming cell CAFC assay is a cell culture-based empirical assay. One such method uses a signature of SLAM family cell surface molecules. Fibrous joint Cartilaginous joint Synovial joint.
Commons category link is on Wikidata. It was originally believed that all HSCs were alike in their self-renewal and differentiation abilities.
Between and , the Committee for Clarification of the Nomenclature of Cells and Diseases of the Blood and Blood-forming Organs issued reports on the nomenclature of blood cells. However, the following nomenclature seems to be, at present, the most prevalent:.
Myeloid tissue can also be present in the liver and spleen  in fetuses , and sometimes even in adults as well, which leads to extramedullary hematopoiesis. Also, some of these markers e.
In the case of fetuses and other extramedullary hematopoiesis , HSCs may also settle in the liver or spleen and develop. For spinal cord tissue, see spinal cord.